GLOSSARY ENTRY (DERIVED FROM QUESTION BELOW) | ||||||
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14:26 Jan 28, 2006 |
French to English translations [PRO] Medical - Medical (general) | |||||||
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| Selected response from: Dr Sue Levy (X) Local time: 10:49 | ||||||
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Summary of answers provided | ||||
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4 +5 | noncardiogenic pulmonary (o)edema |
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5 +1 | pulmonary oedema |
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4 | pulmonary edema due to lesions |
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2 +1 | Wet lung |
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oedeme pulmonaire lesionnel pulmonary oedema Explanation: In answer to your question, the chlorine gas causes pulmonary oedema Inhalation: Initially: irritation of the eyes, nose and throat, followed by coughing and wheezing, dyspnoea, sputum production and chest pain. Larger exposures may lead to hyperchloraemic acidosis; anoxia may lead to cardiac and/or respiratory arrest and pulmonary oedema. Following chemical pneumonitis respiratory distress and chest pain generally subsides within 72 hours; cough may persist for up to 14 days, however in one case reduced airway flow and mild hyopoxemia persisted for 14 months. Reference: http://www.inchem.org |
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oedeme pulmonaire lesionnel Wet lung Explanation: I found this on the GDT site. Not sure though |
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oedeme pulmonaire lesionnel noncardiogenic pulmonary (o)edema Explanation: or "adult respiratory distress syndrome". http://www.emedicine.com/emerg/topic503.htm You will however find "lesional pulmonary oedema" used in English articles, often by non-English authors. http://www.atsdr.cdc.gov/MHMI/mmg172.html Acute exposure to chlorine gas initially causes coughing, eye and nose irritation, lacrimation, and a burning sensation in the chest. Airway constriction and noncardiogenic pulmonary edema may occur. -------------------------------------------------- Note added at 1 hr (2006-01-28 15:44:22 GMT) -------------------------------------------------- From the emedecine.com article: Background: Adult respiratory distress syndrome (ARDS) is a diffuse pulmonary parenchymal injury associated with noncardiogenic pulmonary edema and resulting in severe respiratory distress and hypoxemic respiratory failure. The pathologic hallmark is diffuse alveolar damage (DAD), but lung tissue rarely is available for a pathologic diagnosis. -------------------------------------------------- Note added at 1 hr (2006-01-28 15:47:54 GMT) -------------------------------------------------- To answer your question, it is pulmonary oedema due to lesions of the lung tissue caused by the chlorine (or other toxic substance) -------------------------------------------------- Note added at 1 hr (2006-01-28 16:03:23 GMT) -------------------------------------------------- The "modern" term apparently (I'm an old doctor) is "acute lung injury: http://www.emedmag.com/html/pre/tox/0804.asp In addition, some irritant gases generate reactive oxygen species that are also injurious. Once dissolved, these toxicologic mediators activate pain or irritant receptors and initiate an inflammatory response. They also directly injure the cell membranes of nearby tissue, producing clinical symptoms. In the eyes and oropharynx, this manifests as pain, erythema, injection, or induration; in the distal bronchopulmonary tree, it causes pulmonary capillary leak. The clinicopathologic syndrome associated with capillary leak is ***acute lung injury***, which was formerly termed ***noncardiogenic pulmonary edema***. -------------------------------------------------- Note added at 1 hr (2006-01-28 16:22:12 GMT) -------------------------------------------------- For more synonyms: http://www.fpnotebook.com/LUN67.htm As you see, it's something other than just "pulmonary oedema"! |
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